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N-acetylcysteine chemoprotection without decreased cisplatin antitumor efficacy in pediatric tumor models
DTP cells survive under chemotherapeutic pressure by actively engaging nongenetic adaptive mechanisms, such as CYP3A-mediated drug detoxification, thereby driving adaptive resistance and serving as a key cellular source of treatment failure and tumor recurrence in PDAC
doi:10.1038/s41598-018-22081-7 (PMID 29483571) Ruf S, Rajagopal S, Kadnur SV, et al
however, it is unclear whether intracellular GSH production by tumours themselves is required
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